Dr. Cook Q&A Series: Dr Terry L. Cochran, MD Doctor, Metro Washington DC Area

 Uh, we've seen cases that have been actually resolved. We, we saw one of the, what were the first cases that was, um, misdiagnosed early in December. He had, had a lot of health conditions, had blood pressure, diabetes, um, had, um, RA um, had a lot of joint pain, but he, he was able to bring himself out of it.

Okay. Awesome. Hey, it's, um, director Matt Cook and, uh, we're talking today and it's Friday, March 20th, uh, of the pandemic. And we're excited, uh, because we have Dr. Terry Cochran, uh, with us today. So welcome Terry. Thank you Matt. Great to be here with your audience. Oh yeah. So, um, I'm delighted to have you here maybe, um, we met at a mastermind a couple years ago, and I've super enjoyed, uh, talking to you.

But, um, tell me, tell us a little bit about your practice and then maybe tell me, tell me a little bit about, tell me a little bit about how your day has been today. Okay, well, thank you. Uh, well, I have a clinical practice here in the metro Washington, DC area. And, and though I'm not a doctor, I have a naturopathic doctor that is in my clinic.

And we have, um, we've minted, uh, the Global Sustainable Health Institute, under which my methodology and then my supplements and everything else is, is, um, really underpinned. And so I have developed a, a personalized. Model that looks to seeking to bring the body to sustainable health using your current, uh, your genetic blueprint and your current state of health.

And we've seen thousands and thousands of clients over the last 15 years of practice, and we've had just really, uh, truly efficacious and sustainable results, whether it's Hashimoto's or infertility or MS or working with professional and Olympic athletes. And our week, I was just sharing with Richard, has been, uh, remarkably busy.

So, um, we've actually gone to a virtual consult now just to honor, uh, safe, uh, and responsible citizenship where we can, um, and we'll take in some special cases, but we, we've gone virtually and it, it's been, it's been quite a, quite a busy week. Are you seeing any patients in your practice? Um, we saw, uh, a couple of, uh, autistic patients we saw this week in practice.

We, we really were highly selective as to who we're seeing physically and we're using all, all modes as of practicing safe, uh, interaction. Interesting. So tell me your thoughts. Tell me your thoughts about, uh, Corona and, uh, what, what, what you've seen so far. Have you seen any cases or, or, uh, consulted on any cases over the phone?

Uh, we've seen cases that have been actually resolved. We, we saw one of the, what were the first cases that was, um, misdiagnosed early in December. He was a 67 year old, um, man who'd had, has a lot of, had had a lot of health conditions, had blood pressure, diabetes, um, had, um, RA. Um, had a lot of joint pain, but he, he was able to, um, bri bring himself out of it.

And, um, what we're seeing is any, in his case, what we saw was anything that agitated the nervous system was really, um, contraindicated for him. And so, even post, um, uh, exposure and infection, we're really just looking to calm the system and modulate immunity instead of exacerbating immunity. And we, we do applied kinesiology in my practice and looking at, um, elderberry and astragalus and ashwagandha in his case, were not, they were, they were contraindicated for him.

Oh, really? So that's a, that's a good one. Tell people what, tell people what, um, applied kinesiology is and how you use that technique to determine what supplements to get these home. Of course. So we call it MRI and the wild. Um, so applied kinesiology is a methodology by which, um, a, an individual will hold a vial that carries an electromagnetic signature of anything from garlic to streptococcus or, um, aurelius.

And when we apply pressure to their arm and we complete a circuit between ourselves and the, the patient client, if that vial that has that electro negative signature, uh, has an impairment to the body, then it effectively breaks the circuit and their body, meaning that there is, the nerve conductivity is lost and the nerve cannot fire to the muscle to hold, uh, strength in that arm.

And I'm a, I'm a, a petite woman, and, um, as I say, I, I beat professional athletes up when, um, they can note they have no muscle strength against whatever it is that I'm holding. Uh, they're holding, uh, in their hands and I call it their kryptonite. And it's, it's, we really like about it. It's very efficacious.

Efficacious, and it's real time feedback from the body. Mm-hmm. Yeah, I've, I've actually have quite a bit of experience with, with the, that genre of technique. Which, which, uh, which specific back what, what, what, uh, do you study traditional ak, or is, is, is there a, um, a person that you studied with? I studied, I studied traditional ak, and I can't even remember the gentleman.

Uh, he was the ORI originator, but then I developed my own methodology because I felt that within that model, the body was not giving us enough information. And so, My methodology of AKs, we really look organ to organ and we look at the ground zero, uh, uh, piece of the body or organ of the body or system of the body that then drives and informs everything else.

Okay. So then I'm gonna dig into this one. And this is just kind of interesting for people to sort of follow through, uh, sort of a perspective of how to take care of someone. So you have a case of someone, and he was sensitive neurologically. Did he, did he present with respiratory problems, uh, and pulmonary situations, or did he, did he not have that?

That he did? He did have, he did have pulmonary issues, yes. And then how did the neurological, how lo, how long afterwards did the neurological, uh, uh, expression come. The neurological expression. He, so he presented in late November, early December, and even though he didn't have overt neurological symptomology, vis-a-vis are, are applied kinesiology testing, he was not, he was not, uh, holding, if you will, to anything that would be excitatory.

And we also, we also checked myelin through our AK methodology. And that was, that was still not fully, uh, fully, if you will, um, balanced. And so what we did is we helped support myelination through, um, fossil choline, which is so important for cell membrane and, and the myelin sheath along with vitamin D.

Uh, and he, it was interesting because any form of b12, whether it is methyl cabal with folate or with folic acid or by itself or hydroco, he still failed any, anything that was going to effectively upstart the nervous system. And, and you said he tested negative for you. You said you mentioned three herbs, I think at the beginning that he Oh, yeah.

So, so the, the herbs that, that his body did not like were, and it, it was really interesting because it was an affirmation to some of that, which has been, uh, proffered, um, ashwagandha, astragalus and elderberry. And those three herbs tend to, uh, increase interleukin six. And what we have seen, he also, which is really interesting, Matt, he, he didn't test well against mast cells.

And so that, um, and the prostaglandins one, two, and three. So he failed those. And so what was really interesting there is that it was proving out just empirically, but it was proving out that these herbs that tend to, um, avail the body of interleukin six was, um, in line with, um, a potential histamine slash mast cell slash cytokine response.

So, uh, we found that to be fascinating. So then for people to, for people that are listening, when this, this sort of is to me almost like a little reminiscent of what, how I think about like, complex patients that come in for, for simple cases. It's almost like all you have to think about the common. What's the most known, effective treatment?

So it might be a drug or might be an antioxidant or a vitamin, and the most people will do really great with, with a simple treatment. But for some of the really complex patients, they may, they may not respond to what is a traditional, what's a traditional, well known modality and

right.

The, and so then it's a journey trying to figure out and so what the, what the best is and what people will respond to. And so, um, so that's interesting. And then what, what we know is, is that after people have a pulmonary event, and then particularly in pneumonia, that's when people can start to have this thing where their body starts to drive inflammation.

Yes. And when it drives inflammation, um, if that gets outta control, we call that a cytokine storm. And there's a number of inflammatory cytokines. One is called, uh, interleukin, uh, one one's called TNF alpha. And then, um, I think you probably put tn uh, uh, I six in that category. Mm-hmm. Yes. So kinda interesting.

Any other experiences that you've heard of in terms of talking to people about cases? Well, what we're finding is that, um, depending on, we had a, a case where a, um, a six year old was exposed and it was, we call it C of one. So it's one degree of separation. Now we're, we're, we're calling it C one. C of C of N.

Right. And in her case, she, uh, was exposed and was not tested by the pediatrician. And we just received this feedback today. But she did develop an inflammatory, uh, response, a histamine response, if you will. Now, uh, her, she was having a lot of runny nose and no cough, but she was running a low grade fever.

And so what I find with other viruses, Matt, is that if the body is strong enough, you're, you're, you, you're running at around 99 to a hundred. Not those big spikes. And um, I just found that to be fascinating. Again, empirical. But she may have been exposed, the body is fighting it. Um, and in her case we have her, I see their entire family and her mom is 32 weeks pregnant and they've, they're self quarantined.

So we were very much, uh, looking to, um, make sure that, that, that mom to be, uh, was, was being protected and, and, and, and fully isolated. But I find that was really interesting. She was, she of one, one degree of separation. Her teacher was, um, tested positive and then she came down with, um, what appeared to be respiratory and that low grade fever, which to me doesn't appear to be bacterial.

I usually don't see bacteria as that 99 to a hundred fever. Now did are, were you able to get any testing done? No, the pediatricians wouldn't test for her. They said that she was low risk, unfortunately. Yeah. So that illustrates another problem. We, you know, I, I, I reviewed two or three cases of people who went in, uh, hospitals multiple times and kept getting sent out.

There was one person that went in and outta hospital a couple times, ended up with $35,000 worth of hospital bills and never ended up getting treatment. And so it's, um, and, and, and that probably represented a moderate amount of exposure to the hospital staff. So we're sometimes it feels like we're almost fighting against ourselves in this battle.

Um, so it's been a, it's been a super frustrating sort of experience for me the last couple days. Absolutely. And what we find here, I'm in the metro DC area and we are at. A cherry blossom peak practically. And so pollen is through the roof and it's a very fine line between is it a histamine response from the pollen or are you really actually covid 19?

Because in some cases, pollen will, will elicit flu-like symptoms with a low grade fever. Right? Right. So this one girl in particular hadn't had a history, has not had a history of spring pollen allergies. So it was a departure from her historical, um, profiling and, and how she present presentation rather.

So that's another thing that we need to look at. And we look at it in our, in our practices, how is, and we extrapolate from that because again, this is, this is a disease of extrapolation right now because it's things are changing so quickly. So we have to look at the history of the individual. And if in every other year they've never had any kind of, uh, pollen symptomology, right.

And all of a sudden, It's the same time of year and they're presenting with what I've just shared, then we have to look further. Okay. So then I'll take you up on that. Um, tell me, tell me, let's say somebody comes in with, uh, in the DC metro area and it's a, they, they're allergic to the cherry blossoms and they're having a histamine response.

How does, how do they typically present to you and how's and cause that's interest, that's gonna be interesting for people to hear, hear that side of it. Well, it's really interesting, Matt. What I say is it's gonna go to that person's inflammasome, so inflammasome ORs, or zones of inflammation in the body.

So if your, if your weak spot is. You may change the way that you are, um, uh, experiencing your stools. You may have a runny stool. If you have, um, if your weak spot is, let's say you have ligament laxity, you're gonna be falling out of alignment more and you may have some more joint pain. So this is why, this is why the thing is never the thing you've gotta look at.

Where is that potential person's inflammasome? It can present very differently, more than just, um, I have sneezes and, and I have histamine response because it's really interesting that when we test people through the applied kinesiology, they'll say, oh, I don't have any pollen allergies, but boy, my stomach really hurts.

Or, I don't have any, uh, pollen allergies, but my cervical spine keeps going out of alignment even though I keep getting adjusted every week. So we look to those areas of inflammation or you know, air quote weak points in the body to see is it exacerbated during this period? And then we treat it. Like a pollen sensitivity.

And one of the things that I really like, which is benign, it's a mast cell stabilizer, is chromin and you know, that's an old line mast cell stabilizer. And you can take chromin, and this is by prescription. You can take chromin five, five ml orally or over the counter, which I'm actually really recommending because it, it is inner and benign otherwise is naro, which is, you'll have a chromin spray in, um, in the nose that can actually help modulate again, my goal for this, for this season, whether it's a histamine or a 19 that we really aren't testing, can't test for in our office, it's modulating that that histamine response, which is a part of that larger HPA access.

Ok. So then, um, this, that's really good. So I'm gonna dig into a couple things you said. Tell me what, tell me your definition of inflammasome. Cause that's a, that's a good word. Ok. So I did not make it up, although I, I make up words all the time, but I did not make up, uh, my inflammasome are zones of inflammation in the body that where you have an inflammatory cascade cascade in a specific area of the body, not, it is not systemic.

Right. Right. And so you can have an inflammasome that is, for example, the, the cornea of your eye. Or you can have an inflammasome that is in the, um, uh, in your wrist, right. It can be, it can be joint specific, it can be organ specific, it can be musculoskeletal specific. So then this is a good one. And this I think introduces maybe an interesting idea for people to begin to hear, which is, is that in, when inflammation starts to get out of control, that can happen from all kinds of processes that we're exposed to, like every day or every year.

As an example, cherry blossoms can create a little bit of an overreaction where our body is, becomes a little too sensitive and then has this hyper response. And one of the cells that can have that hyper response is, is called a mast cell. So, which is one of the cells in the body. And so then, uh, what Terry's talking about is a technique to calm that mast cell down with the chromelin.

Right, right, exactly. One. Um, do you think that that would have a, a any benefit? Have you have you in in in Covid? I believe it. I believe it will. Again, you know, there's no one magic bullet. I know, although we're looking at Plaquenil and, uh, uh, azithromycin as, as kind of the, the, the drug of, of, of impact, A positive impact potentially for this, um, for this virus.

But my whole philosophy around how do we manage this is a broad spectrum down regulation of any mass cell or histamine response. Because we know that this little sucker likes mast cells and histamines, which goes into a cytokine storm. So anything that we can do to modulate that is a step in our favor.

Right. I've, which tota, you know, so we're looking at, which also goes to high histamine foods. We've gotta be careful what are, what are the highest histamine foods? Yeah. Yeah. So Aer a sprouted or fermented foods or high histamine. And I will tell you that, um, probiotics are high histamine, yogurt is antihistamine.

So I'm actually telling my folks, unless there's a really big need, and I call it the hierarchy of needs, unless there's a hierarchy of needs in the body for taking a probiotic right now, let's not, because mm-hmm. It can't elicit a histamine response and that's not what we want. Instead, take a non fos probiotic, a prebiotic, like an as, a asparagus or an artichoke.

Um, that's also not going to be a saccharide because we know that sugar will also increase speed of any virus. Ok. So then this is a good one because I think it illustrates. Philosophical differences. Uh, cause last night, last night, we were talking about probiotics with jj. Yes. And so the, the, but there was a logic that we were talking about, and then part of that was, well, if you're doing rectal ozone and up upsetting maybe the microbiome in the gut, yes, then maybe it would be great idea to get probiotics in that setting.

Um, but then we were also generally talking about improving gastrointestinal health as a strategy to, uh, make the body more resilient and le and less susceptible to a gastrointestinal, uh, infection of, of the virus. Now then, but what Terry's talking about is a, uh, similar concept, which is, let's make the body really resistant, but the strategy is to limit histamine.

And because, uh, that that has the benefits that she's talking about. Now what, lemme ask you this, do you think that there are some patients that are probably much more sensitive to histamine and so they would really benefit from this advice and others? Absolutely. That probably would be okay. Right?

Absolutely. And this is when we get into genetics. You know, if you've got, if you've got the GAD gene that looks to histamine and insulin and, and, uh, conversion to glutamate, which is excitatory, or if you have the CMT gene, which goes to dopamine regulation of which in those neurotransmitters, histamine can be one.

If you've got a lot of those excitatory genes and they're expressing as such, then you go, okay, maybe not for this person. And again, in terms of what I call healing and sealing the gut, and I'm very probio, I'm pro probiotic. I don't wanna say that I'm not. But in order to heal and seal the gut, one of the things that I'm availing or I'm recommending is it's.

And in this case it's an antihistamine. And I just read a study, um, last, a couple of nights ago that it also helps down-regulate oxalate crystals. And what we know about oxilates is oxilates tend to be biofilm and biofilm is important. When we look at that secondary complication of pneumonia, which is mycoplasic, which is like a biofilm.

And so we don't wanna do anything that's going to elicit the formation of biofilm and asper Gillis will we know does that. And if we have a dysbiotic gut and we are feeding a lot of probiotics into it with those genetics and those predispositions in a high oxalate load, then we've gotta be really careful cuz you're, we're creating biofilm.

And my research and studies show that biofilm will feed amyloids and amyloids or truncated protein structures that increase the virulence of any virus. Now I don't know about Covid 19 because this is a novel virus, but we've seen it. All the other and the herpes family of viruses. Okay. Terry, you just passed your functional medicine oral boards,

so then that's good. So then, um, let's, let's, let's talk about that because I think that's quite, I, I like, uh, conceptually how you're talking because if I can translate a little bit of that, um, there's, first of all, there's genetic influences, but then what happens in the gut, I you're saying, can have a, a very profound effect on what's happening everywhere else.

And, and that can start, if there's a biofilm that has, let's say, yeast, Or mold or um, or parasites or some combination of those. And then that can be like a kinda somewhat something of a forward base of operations that a virus could infect and then move on from. Is, would, is that fair to say? Fair to say.

And actually our, the, the studies that are out there show that it's really interesting, Matt, I call it the ping pong effect. So biofilm tends to lead to the formation of amyloids. Amyloids are these truncated protein structures. And as you, as, as you know, amyloids are homeostatic device in the body, it's inflammatory response, but it's homeostatic.

But one of the things have found is that supply influencing the amyloid load beyond its homeostatic place, actually feeding viral loads. Amyloids from the food supply feed, the viruses, the, the viruses then go in and hide the inside. The biofilm. The biofilm makes amyloids, which then feeds the viruses.

And so it becomes this circular, um, enforcement against our favor. And so the way I look at it is anything that can create an amyloid load beyond a homeostatic state, anything that feeds biofilm, either from a genetic perspective or from a dietary perspective, is influenced in a, the, an increased viral, uh, virulence.

And so what we tend to do is, and what I try to do with my, uh, clients, is to uncover unpeel back. What are their genetic tendencies? What are they experiencing? What is their organic acid test tell us along with our AK in-house assessment and their symptoms. And move from there. And again, Cetin being such a a, a nice little antihistamine and bioflavonoid and a a a, a tight junction sealer I'm all in, even though it tends to dow regulate the cytochrome P four, some, some snips of the cytochrome P four 50.

Again, the hierarchy of needs are antihistamine and tight junction wins the day. Okay, well, I'd like to say that I likein more than anybody west of the Mississippi, but I'm, I'll give, I'll give you the eastern side of Mississippi since you're on side of the country. Uh, so then, um, so then, um, okay, that's good.

Uh, so then if I can interpret that, and I, and I wanna, don't, don't, lemme forget about the oxalates cuz I'm super interested in that. So. So then this, this kind of thoughtful, comprehensive approach that you're taking is helping you try to get a handle on which patients might have, let's say, gastrointestinal biofilms.

Because if they have those and then they're either produce the amyloid or, um, their, their, um, uh, there's Amy in the diet, then mm-hmm. That becomes basically a, a risk for potentially a worse infection. Absolutely. It's a feeder system. And so then this is, as I kind of, to, to maybe just echo what I think you're saying, which, and which I think is interesting is that when, when, um, In, in functional medicine.

That's why I was joking. I said, you pass your functional medicine boards in fun, in functional medicine. Kinda one, I always like to say there's all these functional medicine gangs. There's like the gang, there's the peptide gang, the regenerative medicine gang. But the, all this gang in the book was the gang that was basically trying to heal the intestines.

Because there's a philosophy that if you could just do that, you might, that might have enough of impact that it everything. Yep. Indeed. And you know, again, I, I look at, I look at multi multiorgan, uh, co functionality and ensure functionality, but. Because my, my roots, my roots are in nutrition. Um, we gotta look at the food and we gotta look at how the body, you know, the nutrigenomics is how, you know, food influenced gene gene expression.

So I had like five people call me over the, in the last 24 hours and, uh, to tell me to do something about food and what to do in this situation, what foods tend to be high in amyloid. So through our, through our research, Matt and uh, actually we found the research out in the clinical literature. This is out of studies of Cambridge and Japan chicken, I call it the dirty bird.

And, um, they've asprey my friend, we, we've, uh, maligned the bird. But what we have found is that because of the crowding conditions, and this is outta, these are, these are clinical studies that are out there, the crowding conditions of, and the way that we inhumanely treat the animals. It is, Creating these truncated proteins in their tissue, and they're not broken down by cooking methodology.

And so when we ingest them, then we are now ingesting an amyloid burden. And then back to our, our genetic predisposition, if you have the MTHFR gene C6 77 T, which you're gonna be less likely to be a good producer of hydrochloric acid because, and you have, you may be impaired in bile salt synthesis, right?

So, and you're missing methyl donors. So you're not making trimethylglycine, which is a precursor to, to hydrochloric acid, which is necessary in protein digestion. You may potentially be, as I say, scribbled, right? Because you're, you're getting an amyloid feeder system from the food supply and you're not an optimal digester of hydrochloric acid.

And we know that hydrochloric acid, I say, is the key that unlocks enzymes. So you may be producing enzymes, but you don't have sufficient hydrochloric acid. You don't have the key in the do hotel, hotel room, right? So it's really critically important from that perspective as well. So chicken, beef, uh, have been the most studied, and I just hypothesized that any animal that's crowded, uh, is going to contain more amyloid, uh, structures.

And I've developed a whole way of eating called the Wild Italian Diet. And the subtitle to that is Living as Nature intended because these animals weren't just, you know, burst here on the planet with amyloids in their tissues. We've, we've created this phenomenon. Um, and we've also found, and this is through our empirical evidence of the thousands of clients that we've seen that have been, that have availed the, uh, themselves of this protocol that I've developed is that when you go wild being bison and, uh, elk and antelope, the amyloid burden, Empirically is reduced because what we're seeing in Matt, we've seen this through the clinical outcomes where we have an IgG reactivation of let's say an Epstein bar virus that then yield Hashimoto's thyroiditis and they have TPOs in the 500 that when you bring down that amyloid burden and look to their genetic blueprinting and their current state of health and look at that personalized plan for them, we see TPOs return to normal.

And that really 500, uh, IgG, B B V is down to 40 and 30. You know, so we've seen that reduction in that reactivation of that viral load, and we've seen that over and over again in our practice. Oh, okay. That's, um, and, and that was, sorry, somebody walked into the room and I got interrupted and, and that I'm doing like multiple IVs while I'm talking to you, so we'll judge me.

Um, but, so then, um, and that, and that was you, you got those. Decreases from diet, from dietary changes of eliminating, uh, basically factory, factory fed animals. Yes. Dietary changes and then supplementing to, to their blueprinting, to their blue based on, based on the empty and stuff like that. Yes. Mm-hmm. So then I'm so 100% supportive of that.

And it's interesting, like, you know, some people are always asking me like, what is the one best thing that you can do? And so then I basically just kind of like rotate around what I say just because I'm kinda curious to see what kind of reaction I get from people. Because sometimes it's just, it's interesting because I'm almost trying to get a little bit of outta them and, and maybe trigger a change.

Um, but I think that if there was one thing that I could say to America right now, it might be. Don't eat chicken if it's not organic. Absolutely. And don't, and, and don't eat beef if it's not organic. Right. And even then, you know, I'm saying if you can eat something else like bison or, or, um, pheasant or Cornish game hen or, or go to wild fish, that's even better because we know fish has high, has high selenium, and selenium is a precursor to glutathione, which is the master antioxidant.

But according to, um, the, another genius doctor, Dr. Todd o I was speaking to him recently and he shared that, um, glutathione has shown epidemiologically to lower viral loads. So, um, that's a selenium to precursor and it also helps increase the assimilation and, uh, production, natural production of glutathione, which is super important for those individuals that have an outright sulfur allergy because glutathione is sulfur based.

Right. So, um, that's a good one because when I heard that this outbreak was coming like 15 days ago mm-hmm. Or like when I really heard that it was coming, the first thing that I did was, um, literally go online and order 50 pounds of buffalo. There you go. Yay. Yeah. So that was, that was great. Um, now, and so then, so then if I'm you, gi thing a little bit, great topic for people to kinda wrap their head around as I, as I read the cases and hear the stories about, um, you know, people, people who are really, the people who are dying in, in Italy and, and Spain and France.

Most of those people have two or three systemic diseases or problems going on, and, and, and in general, when I hear about those type of cases, I often think that a gastrointestinal problem might be an antecedent to those. And so therefore, and when, when we're dealing with people that might have neurological problems and cardiovascular problems, and maybe one other thing, we often start to do testing in the GI tract because we're trying to optimize it.

Because it may be that that's where just like, just like we know that when someone gets a pneumonia, we were talking about this and the virus starts to amplify in the epithelial cells. Yes. And we also know that if we make those cells more healthy, they're less resistant to getting infected by the virus.

In the same way, if we can make the gastrointestinal tract healthy, then that might actually make us less susceptible. And, and if there's less biofilm and there's all, you know, that would make us potentially less susceptible to any viral situation. And basically your, your, your experience through things as simple as dietary modifications.

Have I have born that out? It sounds like in, in treating viral problems and looking at viral load? Absolutely. I say if we heal and seal the gut, we can eat rocks and extract this minerals. Now obviously that's an exaggeration, but that means we have such integrity in the, the lining of the gut that we will only extract the constituents, which are.

Are able to be then assimilated through the cell membrane. What happens is when the tight junction, I say a tight junction looks like pantyhose and not tight junction looks like, um, it looks like fishnet stockings, right? And so larger molecules will pass through the intestinal barrier and the body goes uhoh, I don't know you.

And then it goes from histamine to antigen to antibody in the, you know, in the, in the end result. Cause the body is looking at something that it cannot assimilate and bring through it into its cell membrane for use. And then, you know, recombining to something. Uh, cuz we're having billions of little,